@stephxie Thanks Stephanie.we did not find perk arm of upr to play a significant role but we did not look at lsc specifically. Role of atf4 in aml metabolism has been previously shown by others see for example https://t.co/eozT1ciidm However never linked t
RT @imsmd: Watching the #AACR22 #cancermetabolism minisymposium? Check out the @CD_AACR paper identifying and characterizing the role of se…
RT @imsmd: Watching the #AACR22 #cancermetabolism minisymposium? Check out the @CD_AACR paper identifying and characterizing the role of se…
RT @imsmd: Watching the #AACR22 #cancermetabolism minisymposium? Check out the @CD_AACR paper identifying and characterizing the role of se…
RT @imsmd: Watching the #AACR22 #cancermetabolism minisymposium? Check out the @CD_AACR paper identifying and characterizing the role of se…
Watching the #AACR22 #cancermetabolism minisymposium? Check out the @CD_AACR paper identifying and characterizing the role of serine biosynthesis in FLT3+ #AML by @StefBPhD, @JohnstoneRicky, and colleagues @PeterMacRes https://t.co/XKqwR5JGp4
RT @CD_AACR: From the June issue: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia. https://t.c…
Fantastic work @StefBPhD @JohnstoneRicky
From the June issue: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia. https://t.co/SXqWMYjFOo @StefBPhD @JohnstoneRicky @JohnstoneLab @PeterMacRes @PeterMacCC @DrKristinBrown https://t.co/gq7yWAUEcC
Watching the #AACR21 session on Metabolic Pathways in Cancer? ICYMI in @CD_AACR - #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia @StefBPhD @DrKristinBrown @JohnstoneRicky @JohnstoneLab @PeterMacCC @UniMelb http
#ICYMI: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia. @StefBPhD @JohnstoneRicky @JohnstoneLab @PeterMacRes @PeterMacCC @UniMelb #metabolism https://t.co/nhhql8yPsy https://t.co/q5HsJye1Dm
RT @NatRevClinOncol: Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid leukaemia: https://t.co/1vPnOxWLMp #…
Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid leukaemia: https://t.co/1vPnOxWLMp #leusm #hemeonc
RT @CD_AACR: Just published #OnlineFirst: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia. @St…
@PrecursorCell @DrKristinBrown @JohnstoneRicky Thanks a lot for your interest! Updated link below🙂: https://t.co/wxVhIiqCvr
RT @CD_AACR: Just published #OnlineFirst: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia. @St…
New article: Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid leukaemia. https://t.co/2Pnucltnw1 #AML #leusm #hematology https://t.co/YTK07G0wb5
RT @ElizSMcKenna: Online now in @CD_AACR: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia @Ste…
RT @CD_AACR: Just published #OnlineFirst: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia. @St…
RT @CD_AACR: Just published #OnlineFirst: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia. @St…
RT @CD_AACR: Just published #OnlineFirst: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia. @St…
Just published #OnlineFirst: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia. @StefBPhD @JohnstoneRicky @JohnstoneLab @PeterMacRes @PeterMacCC @DrKristinBrown @UniMelb #metabolism https://t.co/6VV8zNUWGS https:/
RT @StefBPhD: Delighted to share the first first-author publication of my PhD! We discover that the AML mutation FLT3-ITD promotes serine u…
RT @StefBPhD: Delighted to share the first first-author publication of my PhD! We discover that the AML mutation FLT3-ITD promotes serine u…
RT @JohnstoneRicky: So happy to see this work accepted by @CD_AACR. Brilliant job by @StefBPhD in driving the study and the collaboration w…
RT @StefBPhD: Delighted to share the first first-author publication of my PhD! We discover that the AML mutation FLT3-ITD promotes serine u…
Delighted to share the first first-author publication of my PhD! We discover that the AML mutation FLT3-ITD promotes serine uptake + biosynthesis to drive proliferation. A great collaboration between @DrKristinBrown @JohnstoneRicky labs. Thank you both for
So happy to see this work accepted by @CD_AACR. Brilliant job by @StefBPhD in driving the study and the collaboration with @DrKristinBrown and her lab was essential, fun and educational for a metabolism novice like me 🤣 More to come from @PeterMacRes in 20
My absolute pleasure to have played a small role in this impressive study from @DrKristinBrown and @JohnstoneRicky. Congratulations @StefBPhD on all your hard work!
RT @ElizSMcKenna: Online now in @CD_AACR: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia @Ste…
Check out this really interesting study about serine metabolism in AML from @JohnstoneRicky @StefBPhD @JohnstoneLab and colleagues @PeterMacCC @unimelb 👇🏻
RT @ElizSMcKenna: Online now in @CD_AACR: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia @Ste…
RT @ElizSMcKenna: Online now in @CD_AACR: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia @Ste…
Online now in @CD_AACR: #Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid #leukemia @StefBPhD @JohnstoneRicky @JohnstoneLab @PeterMacRes @PeterMacCC @UniMelb #metabolism https://t.co/Ry7f2smggd
Serine biosynthesis is a metabolic vulnerability in FLT3-ITD-driven acute myeloid leukaemia https://t.co/k728JR7rJh